Sorry, but some book science for the newbies.
From WIKI.
“ABA was originally believed to be involved in
abscission, which is how it received its name. This is now known to be the case only in a small number of plants. ABA-mediated signaling also plays an important part in plant responses to environmental stress and plant pathogens.
[6][7] The plant genes for ABA biosynthesis and sequence of the pathway have been elucidated. Seed germination is inhibited by ABA in antagonism with
gibberellin. ABA also prevents loss of seed dormancy.”
“Abscisic acid is also produced in the
roots in response to decreased soil
water potential (which is associated with dry soil) and other situations in which the plant may be under stress. ABA then translocates to the leaves, where it rapidly alters the osmotic potential of stomatal guard cells, causing them to shrink and
stomata to close. The ABA-induced stomatal closure reduces
transpiration (evaporation of water out of the stomata), thus preventing further water loss from the leaves in times of low water availability.”
Enough of that.
The folks in white robes are all over this very ancient phytohormone. Most of the pathways have been mapped and published. I say most because it remains to be elucidated (figured out) how the hormones behave in concert with other hormones and the effects of circadian accumulations on those same hormones.
Anyhow, the jist of this is try and antagonize the ABA pathways with rootzone exogenous applications of ABA. If all goes well the root guard cells will be as pissed off when they encounter the ABA as the stomatal guard cells are when we use the foliar antagonist
MeJA.
“The fingers need not be crossed” says the stoned alchemist.
tbc